Joshua Boyce, MD

Due their biological potency and capacity for rapid production, lipid mediators of inflammation are ideally situated to promote innate immune responses against invading pathogens. At the same time, dysregulated or sustained production of these same mediators can promote immunopathology observed in common human respiratory diseases such as asthma. My lab focuses on how networks of lipid mediators cooperate to both promote and limit type 2 inflammation in the respiratory tract.